Pitalimatrian Albescentis Encephalopathy

Pitalimatrian Albescentis Encephalopathy (shortened: PAE) is a cerebral disease caused by a prion found in the Pitalinc Forest of Skirato.

Cause
PAE is caused by a prion, a misfolded protein. In this case, it is a misfolded nemfopoctin, a protein found in the Nissl body (or tigroid body) of neurons. The misshapen protein causes the endoplasmic reticulum in the Nissl body to produce a protein called amaskophin. Amaskophin triggers the same effect, causing an exponential growth of the number of amaskophin in the body.

Effects
As the neuron runs out of components to produce amaskophin, it begins to break down other proteins and structures within the cell. Because the amaskophin protein is a clear-white color, when more and more amaskophin are produced and less and less of other substances are inside the cell, the neurons begin to turn a white color, hence the name "albescentis". The removal of proteins from the outer cell membrane will cause for the production of P-Protein-Linked Receptors (PPLR). PPLR will emerge at the surface, providing a signal that labels it as infected with PAE. PPLR also releases endorphins called salic metrapallemian neuropepdides. Salic metrapallemian neuropeptides do not travel very far; when they reach the nearest blood vessel, they attatch to the lining and stay there.

As the amaskophin multiply, they break out of the neuron and begin to invade surrounding cells. This spreads to the brain within twenty-four hours, causing muscle spasms, numbness, paralysis, and eventually death.

Diagnosis
PAE can be easily detected if the victim has any fault with his or her nervous system. Also, medulla oblongata of the brain can be inspected, as the prion usually shows the most severe effects here.

Control
PAE can be stopped by an enzyme called Camestepandit-Z (CaZ). CaZ can be administered via injection, IV, or pill. CaZ will enter the bloodstream until they detect salic metrapallemian neuropeptides. When they do so, the enzyme will exit the blood vessel until it reaches the neuron infected with PAE. There, it will join with the PPLR to break down nemfopoctin proteins. After all of the nemfopoctin proteins are broken down, CaZ will then break down itself, releasing an endorphin named Camestepandit Cidalpeptide (CC). CC will cause cells surrounding the infected neurons to produce proteins and other structures needed in nerve cells, then transport it to them, essentially reviving the neurons.